Prof. Dr. Gillian Bates

Profil

Derzeitige StellungProfessor W-3 und Äquivalente
FachgebietMolekulare Biologie und Physiologie von Nerven- und Gliazellen,Humangenetik,Allgemeine Genetik und funktionelle Genomforschung
KeywordsHuntingtons Disease, Neurodegeneration, Mouse Models, Genetic disease

Aktuelle Kontaktadresse

LandVereinigtes Königreich
OrtLondon
Universität/InstitutionUniversity College London (UCL)
Institut/AbteilungInstitute of Neurology

Gastgeber*innen während der Förderung

Beginn der ersten Förderung01.01.2000

Programm(e)

1999Max Planck-Forschungspreis-Programm

Publikationen (Auswahl)

2002E. Hockly, P.M. Cordery, B. Woodman, A. Mahal, A. van Dellen, C. Blakemore, C.M. Lewis, A.J. Hannan, Gillian Bates: Environmental Enrichment Slows Disease Progression in R6/2 Huntington's Disease Mice. In: Ann. Neurol., 2002, 235-242
2001K. Sathasivam, B. Woodman, A. Mahal, F. Bertaux, E.E. Wanker, D.T. Shima, Gillian Bates: Centrosome disorganization in fibrolast cultures derived from R6/2 Huntington's disease (HD) transgenic mice and HD patients. In: Human Molecular Genetics, 2001, 2425-2435
2001D.L. Smith, R. Portier, B. Woodman, E. Hockly, A. Mahal, W.E. Klunk, X.-J. Li, E. Wanker, K.D. Murray, Gillian Bates: Inhibition of Polyglutamine Aggregation in R6/2 HD Brain Slices-Complex Dose-Response Profiles. In: Neurobiology of Disease, 2001, 1017-1026
2001S. Waelter, E. Scherzinger, R. Hasenbank, E. Nordhoff, R. Lurz, H. Goehler, C. Gauss, K. Sathasivam, Gillian Bates, H. Lehrach, E.E. Wanker: The huntingtin interacting protein HIP1 is a clathrin and alpha-adaptin-binding protein involved in receptor-mediated endocytosis. In: Human Molecular Genetics, 2001, 1807-1817
2000J.S. Steffan, A. Kazantsev, O. Spasic-Boskovic, M. Greenwald, Y.-Z. Zhu, H. Gohler, E.E. Wanker, Gillian Bates, D.E. Housman, L.M. Thompson: The Huntington's disease protein interacts with p53 and CREB-binding protein and represses transcription. In: PINAS, 2000, 6763-6768